Characteristics of hypokalemic paralysis secondary to type 1 renal tubular acidosis in primary sjogren syndrome

LIU Peng; WANG Qian; and PU Chuanqiang

PDF(498 KB)

Medical Journal of the Chinese People Armed Police Forces ›› 2017, Vol. 28 ›› Issue (6) : 614-616.

ORIGINAL ARTICLES

Characteristics of hypokalemic paralysis secondary to type 1 renal tubular acidosis in primary sjogren syndrome

LIU Peng¹,WANG Qian²,and PU Chuanqiang³

Author information +

History +

Abstract

Objective To explore the clinical features of hypokalemic paralysis secondary to type 1 renal tubular acidosis in primary sjogren syndrome.Methods We retrospectively studied eighteen cases of hypokalemic paralysis secondary to type 1 renal tubular acidosis in primary sjogren syndrome.Results The clinical manifestations of hypokalemic paralysis secondary to type 1 renal tubular acidosis preceded the development of sicca syndrome in eleven cases of primary sjogren syndrome. In the other seven cases, the prodromal symptoms were oral dryness (xerostomia) and dryness of eyes (kerato conjunctivitis sicca). All the eighteen cases had hypokalemic paralysis. Among the ten cases of bone disease, nine had osteoporosis, and one had osteomalacia.Conclusions Patients with hypokalemic paralysis secondary to renal tubular acidosis in primary sjogren syndrome have typical clinical features and early diagnosis can lead to proper treatment .

Key words

primary sjogren syndrome / renal tubular acidosis / hypokalemic paralysis

Cite this article

EndNote

Ris (Procite)

Bibtex

Download Citations

LIU Peng,WANG Qian,and PU Chuanqiang. Characteristics of hypokalemic paralysis secondary to type 1 renal tubular acidosis in primary sjogren syndrome[J]. Medical Journal of the Chinese People Armed Police Forces. 2017, 28(6): 614-616

References

[1] Sung C C, Cheng C J, Chiang W F, et al. Etiologic and therapeutic analysis in patients with hypokalemic nonperiodic paralysis[J]. Am J Med, 2015, 128(3): 289-296.
[2] Mounica K J,Sastry K,Reddy S K ,et al. Hypokalemic paralysis as a presenting manifestation of primary Sjogren’s syndrome[J]. J Assoc Physicians India, 2016, 64(1): 123.
[3] Vitali C, Bombardieri S, Jonsson R, et al. Classification criteria for Sjgren’s syndrome: a revised version of the European criteria proposed by the American-European Consensus Group [J]. Ann Rheum Dis, 2002, 61(6): 554-558.
[4] Masanori K , Tetsuki A, Yoshitaka M, et al. Hypokalemic paralysis and osteomalacia secondary to renal tubular acidosis in a case with primary Sjgren’s syndrome [J]. Mod Rheumatol, 2006, 16(1): 48-51.
[5] Duffles-Amarante G B, Zotin M C, Rocha E, et al. Renal tubular dysfunction in patients with primary Sjogren syndrome [J]. Clin Nephrol, 2014 , 81(3): 185-191.
[6] Milford F, Meggan M. Renal tubular acidosis, Sjogren syndrome, and bone disease[J]. Arch Intern Med, 2004, 164(8): 905-909.
[7] Fumi T, Junichi H,Naoki S,et al. Auto antibodies against carbonic anhydrase Ⅱ are increased in renal tubular acidosis associated with Sjgren syndrome [J]. Am J Med, 2005, 118(2): 181-184.
[8] Bogdanovi R, Basta-Jovanovi G, Putnik J, et al. Renal involvement in primary Sjogren syndrome of childhood: case report and literature review [J]. Mod Rheumatol, 2013, 23(1): 182-189.
[9] Khan U F, Tsu L C, Tan K B, et al. Hypokalaemia in Sjgren’s syndrome: the missing piece [J]. Clin Med (Lond), 2017, 17(1): 40-42.