[1] 刘淑珍,罗小军.玫瑰痤疮的中西医研究概况[J].新疆中医药,2019,37(1):127-129.
[2] 李 吉,谢红付.玫瑰痤疮的定义及分型[J].皮肤病与性病,2017,39(2):89-90.
[3] 李 健,袁 超.玫瑰痤疮的病因及流行病学[J].皮肤病与性病,2017,39(2):90-99.
[4] 中国医师协会皮肤科医师分会皮肤美容亚专业委员会.中国玫瑰痤疮诊疗专家共识(2016)[J].中华皮肤科杂志,2017,(3):156-161.
[5] Parisi R, Yiu Z N. The worldwide epidemiology of rosacea[J]. Br J Dermatol,2018,179(2):239-240.
[6] Lee J B,Moon J, Moon K R, et al. Epidemiological and clinical features of rosacea in Korea: A multicenter cross-sectional study[J]. J Dermatol, 2018, 45(5):546-553.
[7] Dlova N C, Mosam A, Tsoka G J. The spectrum and sequelae of acne in black south africans seen in tertiary institutions[J]. Skin Appendage Disord, 2018, 4(4):301-303.
[8] Tan J, Schfer H, Araviiskaia E, et al. Prevalence of rosacea in the general population of Germany and Russia-The RISE study[J]. J Eur Acad Dermatol Venereol, 2016, 30(3):428-434.
[9] 李 健,郝 飞.玫瑰痤疮发病机制的研究进展[J].国际皮肤性病学杂志,2015,(1):15-18.
[10] Palleschi G M,Torchia D. Rosacea in a monozygotic twin[J]. Australas J Dermatol,2007, 48(2):132-133.
[11] Aldrich N, Gerstenblith M, Fu P, et al. Genetic vs environmental factors that correlate with rosacea: a cohort-based survey of twins[J]. Jama Dermat, 2015 , 151(11):1213-1219.
[12] Ozlu E,Karadag A S, Ozkanli S, et al. Comparison of TLR-2, TLR-4, and antimicrobial peptide levels in different lesions of acne vulgaris[J].Cutan Ocul Toxicol, 2016 , 35(4):300-309.
[13] 纪永佳,陈彦香.维生素D免疫调节作用与抗菌肽作用的研究进展[J].当代医学,2018,24(6):166-169.
[14] Steinhoff M,Vocanson M,Voegel J J, et al. Topical Ivermectin 10 mg/g and oral doxycycline 40 mg modified-release: current evidence on the complementary use of anti-Inflammatory rosacea treatments[J]. Adv Ther, 2016, 33(9):1481-1501.
[15] Mascarenhas N L,Wang Z,Chang Y L, et al. TRPV4 mediates mast cell activation in cathelicidin-induced rosacea Inflammation[J]. J Invest Dermatol, 2017, 137(4):972-975.
[16] Peter R, Hansen M D, Gunnar H, et al. Patients with rosacea have increased risk of dementia[J]. Ann Neurol, 2016, 79(6):921-928.
[17] Steinhoff M,Schmelz M,Schauber J. Facial erythema of rosacea-aetiology, different pathophysiologies and treatment options[J]. Acta Derm Venereol, 2016, 96(5):579-586.
[18] Tsiskarishvili T,Katsitadze A,Tsiskarishvili N V, et al. Features rosacea pathoenesis in perimenopausal women[J]. Eorgian Math J, 2018, (282):99-102.
[19] Yang Y, Wang H, Kouadir M, et al. Recent advances in the mechanisms of NLRP3 inflammasome activation and its inhibitors[J]. Disease, 2019, 10(2):128.
[20] Stephan A, Batinica M, Steiger J, et al. LL37:DNA complexes provide antimicrobial activity against intracellular bacteria in human macrophages[J]. Immunology,2016, 148(4):420-432.
[21] Kim M, Kim K E, Jung H Y, et al. Recombinant erythroid differentiation regulator 1 inhibits both inflammation and angiogenesis in a mouse model of rosacea[J].Exp Dermatolt, 2015, 24(9):680-685.
[22] 李 婷,胡 阳.LL-37在玫瑰痤疮发病机制中的研究进展[J].中国皮肤性病学杂志,2015,29(07):738-740.
[23] Muto Y, Wang Z, Vanderberghe M, et al. Mast cells are key mediators of cathelicidin-initiated skin inflammation in rosacea[J]. J Invest Dermatol, 2019, 134(11):2728-2736.
[24] Schwab V D, Sulk M, Seeliger S, et al. Neurovascular and neuroimmune aspects in the pathophysiology of rosacea[J]. J Invest Dermatol, 2011, 15(1):53-62.
[25] Karpouzis A,Avgeridis P, Tripsianis G, et al. Assessment of tachykinin receptor 3’gene polymorphism rs3733631 in rosacea[J]. Int Sch Res Notices,2015, 4(3):402.
[26] Anne L S, Chang, Inbar R, et al.Researchers from stanford university and 23andme discover genetic links to rosacea[J]. J Invest Dermatol,2015, 135(6):1548-1555.
[27] Woo Y R, Kim J S, Lim J H, et al. Acquired diffuse slate-grey facial dyspigmentation due to henna: an unrecognized cause of pigment contact dermatitis in Korean patients[J]. Eur J Dermatol, 2018, 28(5):644-648.
[28] Aponte J L, Chiano M N, Yerges L M, et al. Assessment of rosacea symptom severity by genome-wide association study and expression analysis highlights immuno-inflammatory and skin pigmentation genes[J].Hum Mol Genet, 2018, 30:2762-2772.
[29] Shen X B, Wang J, Li P F, et al. Screening of susceptibility genes and multi-gene risk analysis in gastric cancer[J]. Med Oncol, 2014, 31(10):196.
[30] Egenerg A, Hansen P R, Gislason G H, et al.Exploring the Association between rosacea and parkinson disease:a danish nationwide cohort study[J].JAMA neurology,2016,73(5):529-534.
[31] Muller M D,Sauder C L,Ray C A. Melatonin attenuates the skin sympathetic nerve response to mental stress[J]. Am J Physiol Heart Circ Physiol,2013, 305(9): 1382-1386.
[32] Wilson T E, Toma K, Metzler-Wilson K, et al.Augmented supraorbital skin sympathetic nerve activity responses to symptom trigger events in rosacea patients[J]. FASEB J,2012,26:1092-1110.
[33] Verena D. Schwab, Mathias Sulk, et al. Neurovascular and neuroimmune aspects in the pathophysiology of rosacea[J].J Investig Dermatol Symp Proc, 2011, 15(1):53-62.
[34] Schwab V D, Sulk M, Seeliger S, et al.Neurovascular and neuroimmune aspects in the pathophysiology of rosacea[J]. J Investig Dermatol Symp Proc,2011,15(1):53-60.
[35] Christensen C E, Andersen F S, Wienholtz N, et al. The relationship between migraine and rosacea: systematic review and meta-analysis[J].Cephalalgia, 2018, 38(7):1387-1398.
[36] Alinia H, Cardwell L A, Tuchayi S M, et al. Screening for depression in rosacea patients[J].Cutis, 2018, 102(1):36-38.
[37] Kim B Y, Moon H R, Ryu H J. Comparative efficacy of short-pulsed intense pulsed light and pulsed dye laser to treat rosacea[J]. J Cosmet Laser Ther,2018,4:1-46.
[38] 陈 伟,马晓燕,侯海利. 中西医结合疗法治疗重度痤疮的疗效观察[J]. 武警医学,2017,28(2):191-194.
[39] Kallis P J, Price A, Dosal J R, et al. A biologically based approach to acne and rosacea[J].J Drugs Dermatol, 2018, 17(6):611-617.