[1] Shih K C, Lam S L, Tong L. A systematic review on the impact of diabetes mellitus on the ocular surface[J]. Nutrition & Diabetes, 2017, 7(3):e251. [2] 毛滨生,刘 燕,吴小苏, 等.74例2型糖尿病并发症死因分析[J].武警医学, 2000, 11(7):401-402. [3] 杨慧芳,魏碧玉,马丽娟.糖尿病周围神经病变常见危险因素研究进展[J].武警医学, 2020, 31(9):819-822. [4] Parim B, Sathibabu V V, Saravanan G. Diabetic cardiomyopathy: molecular mechanisms, detrimental effects of conventional treatment, and beneficial effects of natural therapy[J]. Heart Fail Rev,2019, 24(2):279-299. [5] Prabhu M, Yaxuan L, David K, et al. Angiogenic mechanisms of Human CD34+ stem cell exosomes in the repair of ischemic hindlimb[J]. Circ Res, 2017, 120(9): 1466-1476. [6] Xie F, Zhou X, Fang M, et al. Extracellular vesicles in cancer immune microenvironment and cancer immunotherapy[J]. Adv Sci (Weinh), 2019, 6(24):. [7] Hathaway Q A, Pinti M V, Durr A J, et al. Regulating microRNA expression: at the heart of diabetes mellitus and the mitochondrion[J]. Am J Physiol Heart Circ Physiol, 2018, 314(2): H293-H310. [8] Bellin G, Gardin C, Ferroni L, et al. Exosome in cardiovascular diseases: a complex world full of hope[J]. Cells, 2019, 8(2):166. [9] 武庆娟, 陈恒文, 高 健, 等. 外泌体miRNA在心血管疾病中的研究进展[J].中国病理生理杂志, 2020,12(2):371-377. [10] Jia G, Hill M A, Sowers J R. Diabetic cardiomyopathy: an update of mechanisms contributing to this clinical entity[J]. Circ Res, 2018, 122(4):624-638. [11] Adlakha Y K, Seth P. The expanding horizon of miRNAs in cellular reprogramming[J]. Prog Neurobiol, 2017, 148:21-39. [12] Xue R, Tan W, Wu Y, et al. Role of exosomal miRNAs in heart failure[J]. Front Cardiovasc Med, 2020, 7:592412. [13] Jia G, Whaley C A,Sowers J R. Diabetic cardiomyopathy: a hyperglycaemia- and insulin-resistance-induced heart disease[J]. Diabetologia, 2018, 61(1): 21-28. [14] Nirmala K, Abdoh T,Zhou J F, et al. Pharmacological strategies to lower crosstalk between nicotinamide adenine dinucleotide phosphate (NADPH) oxidase and mitochondria[J]. Biomed Pharmacother, 2019, 111: 1478-1498. [15] Samarjit D, Mark K, Brittany D E, et al. Divergent effects of mir-181 family members on myocardial function through protective cytosolic and detrimental mitochondrial miRNA targets[J]. J Am Heart Assoc, 2017, 6(3): e004694. [16] Azzouzi H, Leptidis S, Dirkx E, et al. The hypoxia-inducible microRNA cluster miR-199a~214 targets myocardial PPARδ and impairs mitochondrial fatty acid oxidation[J].Cell Metab, 2013, 18(3):341-354. [17] Singh R M, Waqar T, Howarth F C, et al. Hyperglycemia-induced cardiac contractile dysfunction in the diabetic heart[J]. Heart Fail Rev, 2018, 23(1): 37-54. [18] Chen C, Yang S L, Li H P, et al. Mir30c is involved in diabetic cardiomyopathy through regulation of cardiac autophagy via BECN1[J]. Mol Ther Nucleic Acids, 2017, 7(C): 127-139. [19] Zhang W, Xu W T,Feng Y, et al. Non-coding RNA involvement in the pathogenesis of diabetic cardiomyopathy[J]. J Cell Mol Med, 2019, 23(9): 5859-5867. [20] Feng B, Chen S L, George B, et al. Mir133a regulates cardiomyocyte hypertrophy in diabetes[J]. Diabetes Metab Res Rev, 2010, 26(1): 40-49. [21] Ma Q, Ma Y, Wang X N, et al. Circulating mir-1 as a potential predictor of left ventricular remodeling following acute st-segment myocardial infarction using cardiac magnetic resonance[J]. Quant Imaging Med Surg, 2020, 10(7): 1490-1503. [22] Yu F, Chapman S, Pham D L, et al. Decreased mir-150 in obesity-associated type 2 diabetic mice increases intraocular inflammation and exacerbates retinal dysfunction[J]. BMJ Open Diabetes Res Care, 2020, 8(1):e001446. [23] Gabriela P D, Takano A P, Maria L M. MiRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy-role of type 1 nngiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation[J]. Mol Cell Endocrinol, 2013, 374(1-2): 117-124. [24] Raut S K, Singh G B, Rastogi B, et al. MiR-30c and mir-181a synergistically modulate p53-p21 pathway in diabetes induced cardiac hypertrophy[J]. Mol Cell Biochem, 2016, 417(1-2):191-203. [25] Wei H, Bu R,Yang Q H, et al. Exendin-4 protects against hyperglycemia-induced cardiomyocyte pyroptosis via the AMPK-TXNIP pathway[J]. J Diabetes Res, 2019, 2019:8905917. [26] Lee S, Suh G Y,Ryter S W, et al. Regulation and function of the nucleotide binding domain leucine-rich repeat-containing receptor, pyrin domain-containing-3 inflammasome in lung disease[J]. Am J Respir Cell Mol Biol, 2016, 54(2): 151-160. [27] Zhu Y, Qian X,Li J, et al. Astragaloside-Ⅳ protects H9C2(2-1) cardiomyocytes from high glucose-induced injury via miR-34a-mediated autophagy pathway[J]. Artificial Cells, 2019, 47(1): 4172-4181. [28] Yu X Y, Song Y H, Geng Y J, et al. Glucose induces apoptosis of cardiomyocytes via microRNA-1 and IGF-1[J]. BBRC, 2008, 376(3):548-552. [29] Delfan M, Delphan M,Kordi M R, et al. High intensity interval training improves diabetic cardiomyopathy via miR-1 dependent suppression of cardiomyocyte apoptosis in diabetic rats[J]. J Diabetes Metab Disord, 2020, 19(1): 145-152. [30] Nilanjan G, Rajesh K. Molecular mechanism of diabetic cardiomyopathy and modulation of microRNA function by synthetic oligonucleotides[J]. Cardiovasc Diabetol, 2018, 17(1): 43. [31] Feng B, Cao Y,Chen S L, et al. MiR-200b mediates endothelial-to-mesenchymal transition in diabetic cardiomyopathy[J]. Diabetes, 2016, 65(3): 768-779. [32] Li X, Meng C,Han F, et al. Vildagliptin attenuates myocardial dysfunction and restores autophagy via miR-21/SPRY1/ERK in diabetic mice heart[J]. Front Pharmacol, 2021,12:634365. [33] Eva V R, Lillian B S,Jeffrey E T, et al. Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 incardiac fibrosis[J]. Proc Natl Acad Sci.U S A, 2008, 105(35): 13027-13032. [34] Jane C, Hana F, Bing Y X, et al. PARP mediates structural alterations in diabetic cardiomyopathy[J]. J Mol Cell Cardiol, 2008, 45(3): 385-393. [35] Lee W S, Kim J. Application of animal models in diabetic cardiomyopathy[J]. Diabetes Metab J,2021,45(2):129-145.