重症恶性疟疾伴急性心肌炎1例

刘新林, 刘冀琴, 王娜

武警医学 ›› 2022, Vol. 33 ›› Issue (9) : 783-785.

PDF(1557 KB)
PDF(1557 KB)
武警医学 ›› 2022, Vol. 33 ›› Issue (9) : 783-785.
临床经验总结

重症恶性疟疾伴急性心肌炎1例

  • 刘新林1, 刘冀琴2, 王娜3
作者信息 +
文章历史 +

引用本文

导出引用
刘新林, 刘冀琴, 王娜. 重症恶性疟疾伴急性心肌炎1例[J]. 武警医学. 2022, 33(9): 783-785
中图分类号: R541.9   

参考文献

[1] 张佳欣. 中国正式获世卫组织消除疟疾认证[N]. 科技日报, 2021-07-05.
[2] Franzen D, Curtius J M, Heitz W, et al. Cardiac involvement during and after malaria [J]. Clin Investig, 1992, 70(8): 670-673.
[3] Farombi E O, Shyntum Y Y, Emerole G O. Influence of chloroquine treatment and Plasmodium falciparum malaria infection on some enzymatic and non-enzymatic antioxidant defense indices in humans [J]. Drug Chem Toxicol, 2003, 26(1): 59-71.
[4] Wennicke K, Grockiego F, Wichmann D, et al. Glycosylphosphatidylinositol-induced cardiac myocyte death might contribute to the fatal outcome of Plasmodium falciparum malaria [J]. Apoptosis, 2008, 13(7): 857-866.
[5] Ralli S, Horwich T B, Fonarow G C. Relationship between anemia, cardiac troponin I, and B-type natriuretic peptide levels and mortality in patients with advanced heart failure [J]. Am Heart J, 2005, 150(6): 1220-1227.
[6] Ehrhardt S, Mockenhaupt F P, Anemana S D, et al. High levels of circulating cardiac proteins indicate cardiac impairment in african children with severe Plasmodium falciparum malaria [J]. Microbes Infect, 2005, 7(11-12): 1204-1210.
[7] Bhat S, Alva J, Muralidhara K, et al. Malaria and the heart [J]. BMJ Case Rep, 2012, 2012.
[8] Pino P, Vouldoukis I, Kolb J P, et al. Plasmodium falciparum--infected erythrocyte adhesion induces caspase activation and apoptosis in human endothelial cells [J]. J Infect Dis, 2003, 187(8): 1283-1290.
[9] White N J. Loading dose in antimalarial prophylaxis[J]. Trans R Soc Trop Med Hyg, 1985, 79(5): 742-742.
[10] Gupta R K, Van Vugt M, Paiphun L, et al. Short report: no evidence of cardiotoxicity of atovaquone-proguanil alone or in combination with artesunate [J]. Am J Trop Med Hyg, 2005, 73(2): 267-268.
[11] Kelman S N, Sullivan S G, Stern A. Stimulation of ATP hydrolysis by chloroquine and primaquine in human red blood cells [J]. Biochem Med, 1983, 29(3): 379-384.
[12] Marrelli M T, Brotto M. The effect of malaria and anti-malarial drugs on skeletal and cardiac muscles [J]. Malar J, 2016, 15(1): 524.
[13] Chaulin A M, Duplyakova P D, Duplyakov D V. Circadian rhythms of cardiac troponins: mechanisms and clinical significance[J]. Russian Journal of Cardiology, 2020, 25(3S): 4061.
[14] Chaulin A M, Duplyakov D V. High-sensitivity cardiac troponins: circadian rhythms[J]. Cardiovascular Therapy and Prevention (Russian Federation), 2021, 20(1):2639.
[15] Chauin A. The main causes and mechanisms of increase in cardiac troponin concentrations other than acute myocardial infarction (Part 1): physical exertion, inflammatory heart disease, pulmonary embolism, renal failure, sepsis [J]. Vasc Health Risk Manag, 2021, 17: 601-617.
[16] Wilhelm J, Hettwer S, Schuermann M, et al. Elevated troponin in septic patients in the emergency department: frequency, causes, and prognostic implications [J]. Clin Res Cardiol, 2014, 103(7): 561-567.
[17] Ali S A, Kazmi S, Jalal M, et al. Frequency of elevated troponin T in patients of hronic renal failure without clinically suspected acute myocardial infarction [J]. J Ayub Med Coll Abbottabad, 2019, 31(3): 364-367.

PDF(1557 KB)

Accesses

Citation

Detail

段落导航
相关文章

/